News & Announcements
Read the latest news from Northwestern University Feinberg School of Medicine’s Department of Cell & Developmental Biology. The links below take you to articles where you can learn more about our faculty’s latest achievements, awards and honors.
A novel combination treatment may increase the ability of monoclonal antibodies to control viral infection in patients diagnosed with HIV, according to a Northwestern Medicine study.
An international collaboration for which Luisa Iruela-Arispe, PhD, chair of Cell and Developmental Biology, serves as North American coordinator, has received a five-year, $7 million Transatlantic Networks of Excellence Program award from the Leducq Foundation.
Northwestern Medicine investigators have discovered that a subset of proteins in mitochondria of brain and heart cells are long-lived, supporting the long-term stability of mitochondrial complex architecture.
Migrating cells use stiffened microtubules to push through tissue barriers, seeking out weak points in tissue, according to a recent study.
A study led by Northwestern Medicine investigators has identified the molecular mechanisms within protein complexes that promote cell-to-cell adhesion and communication.
A protein helps direct the flow of materials in the Drosophila ovary during the development of egg cells, according to a Northwestern Medicine study.
Enhancing autophagy in fat could help treat diabetes, according to a Northwestern Medicine study published in Cell Reports.
Northwestern Medicine investigators discovered that nuclear surface proteins called nuclear lamin isoforms regulate nuclear pore complexes, shedding light on the role molecular mechanisms play in genetic mutations behind many rare diseases.
Northwestern Medicine investigators have discovered that inhibiting a specific autophagy protein found in dopaminergic neurons may prevent reward-related behaviors induced by cocaine misuse.
Feinberg investigators are breaking down the mechanisms of aging and designing solutions to extend healthy living.
A rare genetic mutation found in patients with Alzheimer’s may provide further insight into the pathologic mechanisms that cause the disease.